part Section I: Oxidative Stress, Metabolic Syndrome, Obesity, Diabetes, and Uncoupling Proteins -- chapter 1 The Metabolic Syndrome Defined -- chapter 2 The Metabolic Syndrome: The Question of Balance between the Pro-Inflammatory Effect of Macronutrients and the Anti-Inflammatory Effect of Insulin -- chapter 3 The Role of Oxidative Stress in Diseases Associated with Overweight and Obesity -- chapter 4 Metabolic Syndrome Due to Early Life Nutritional Modifications -- chapter 5 Oxidative Stress and Antioxidants in the Perinatal Period -- chapter 6 Maternal Obesity, Glucose Intolerance, and Inflammation in Pregnancy -- chapter 7 Obesity, Nutrigenomics, Metabolic Syndrome, and Type 2 Diabetes -- chapter 8 Post-Prandial Endothelial Dysfunction, Oxidative Stress, and Inflammation in Type 2 Diabetes -- chapter 9 Obesity and Inflammation: Implications for Atherosclerosis -- chapter 10 Oligomeric Composition of Adiponectin and Obesity -- chapter 11 Insulin-Stimulated Reactive Oxygen Species and Insulin Signal Transduction -- chapter 12 Intracellular Signaling Pathways and Peroxisome Profiliferator-Activated Receptors in Vascular Health in Hypertension and in Diabetes -- chapter 13 Role of Uncoupling Protein 2 in Pancreatic Cell Function: Secretion and Survival -- part Section II: Influence of Dietary Factors, Micronutrients, and Metabolism -- chapter 14 Nutritional Modulation of Inflammation in Metabolic Syndrome -- chapter 15 Dietary Fatty Acids and Metabolic Syndrome -- chapter 16 Lipid-Induced Death of Macrophages: Implication for Destabilization of Atherosclerotic Plaques -- chapter 17 -Lipoic Acid Prevents Diabetes Mellitus and EndothelialDysfunction in Diabetes-Prone Obese Rats -- chapter 18 Lipoic Acid Blocks Obesity through Reduced Food Intake, Enhanced Energy Expenditure, and Inhibited Adipocyte Differentiation -- chapter 19 Effects of Conjugated Linoleic Acid and Lipoic Acid on Insulin Action in Insulin-Resistant Obese Zucker Rats -- chapter 20 Trivalent Chromium Supplementation Inhibits Oxidative Stress, Protein Glycosylation, and Vascular Inflammation in High Glucose-Exposed Human Erythrocytes and Monocytes.